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Congenital Heart Defect v0.516 ABL1 Zornitza Stark Phenotypes for gene: ABL1 were changed from Congenital heart defects and skeletal malformations syndrome (MIM# 617602) to Congenital heart defects and skeletal malformations syndrome (MIM# 617602); Human ABL1 Deficiency Syndrome (HADS)
Congenital Heart Defect v0.515 ABL1 Zornitza Stark Publications for gene: ABL1 were set to PMID: 28288113
Congenital Heart Defect v0.514 ABL1 Zornitza Stark Mode of inheritance for gene: ABL1 was changed from MONOALLELIC, autosomal or pseudoautosomal, imprinted status unknown to BOTH monoallelic and biallelic, autosomal or pseudoautosomal
Congenital Heart Defect v0.513 ABL1 Zornitza Stark changed review comment from: New gene-disease association - 3 consanguineous families reported.

PMID: 39155385 - Lebanese consanguineous family with a history of cardiac abnormalities. Homozygous LoF variant (NM_007313.3:c.1A > G-p.Met1?) was identified in sequencing and was shown to only be expressed in one isoform of ABL1.

PMID: 38743093 - two other consanguineous families presenting with congenital malformations and facial dysmorphism. Homozygous LoF variants were identified - Gly671Alafs*93 and Glu675Glyfs*71 (both absent from gnomAD v4.1); to: New gene-disease association - 3 consanguineous families reported.

PMID: 39155385 - Lebanese consanguineous family with a history of cardiac abnormalities. Homozygous LoF variant (NM_007313.3:c.1A > G-p.Met1?) was identified in sequencing and was shown to only be expressed in one isoform of ABL1.

PMID: 38743093 - two other consanguineous families presenting with congenital malformations and facial dysmorphism. Homozygous LoF variants were identified - Gly671Alafs*93 and Glu675Glyfs*71 (both absent from gnomAD v4.1)

Postulated that LOF variants affecting solely ABL1 isoform 1b may lead to the distinct autosomal recessive new phenotype
Congenital Heart Defect v0.513 ABL1 Zornitza Stark reviewed gene: ABL1: Rating: AMBER; Mode of pathogenicity: None; Publications: 39155385, 38743093; Phenotypes: Human ABL1 Deficiency Syndrome (HADS); Mode of inheritance: BIALLELIC, autosomal or pseudoautosomal
Congenital Heart Defect v0.49 ABL1 Zornitza Stark Marked gene: ABL1 as ready
Congenital Heart Defect v0.49 ABL1 Zornitza Stark Gene: abl1 has been classified as Green List (High Evidence).
Congenital Heart Defect v0.49 ABL1 Zornitza Stark Classified gene: ABL1 as Green List (high evidence)
Congenital Heart Defect v0.49 ABL1 Zornitza Stark Gene: abl1 has been classified as Green List (High Evidence).
Congenital Heart Defect v0.48 ABL1 Ain Roesley gene: ABL1 was added
gene: ABL1 was added to Congenital Heart Defect. Sources: Literature
Mode of inheritance for gene: ABL1 was set to MONOALLELIC, autosomal or pseudoautosomal, imprinted status unknown
Publications for gene: ABL1 were set to PMID: 28288113
Phenotypes for gene: ABL1 were set to Congenital heart defects and skeletal malformations syndrome (MIM# 617602)
Penetrance for gene: ABL1 were set to unknown
Review for gene: ABL1 was set to GREEN
Added comment: PMID: 28288113: six affected individuals from 4 unrelated families who shared similar clinical features including dysmorphic facial features (6/6), congenital heart disease (CHD, 6/6), skeletal abnormalities (6/6), joint problems (5/6), failure to thrive (5/6), gastrointestinal problems (5/6), and male genital/sexual abnormalities (3/4). Missense variants with 3 families sharing the same variant (Tyr245Cys).
Authors also noted similar congenital malformations observed in fetuses exposed to the selective tyrosine kinase inhibitor imatinib, and patients with constitutional ABL1 variants
Sources: Literature