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Mendeliome v1.4873 MDGA1 Chirag Patel Classified gene: MDGA1 as Amber List (moderate evidence)
Mendeliome v1.4873 MDGA1 Chirag Patel Gene: mdga1 has been classified as Amber List (Moderate Evidence).
Mendeliome v1.4873 MDGA1 Chirag Patel Classified gene: MDGA1 as Amber List (moderate evidence)
Mendeliome v1.4873 MDGA1 Chirag Patel Gene: mdga1 has been classified as Amber List (Moderate Evidence).
Mendeliome v1.4873 MDGA1 Chirag Patel Classified gene: MDGA1 as Amber List (moderate evidence)
Mendeliome v1.4873 MDGA1 Chirag Patel Gene: mdga1 has been classified as Amber List (Moderate Evidence).
Mendeliome v1.4872 MDGA1 Chirag Patel Classified gene: MDGA1 as Amber List (moderate evidence)
Mendeliome v1.4872 MDGA1 Chirag Patel Gene: mdga1 has been classified as Amber List (Moderate Evidence).
Mendeliome v1.4871 MDGA1 Chirag Patel Marked gene: MDGA1 as ready
Mendeliome v1.4871 MDGA1 Chirag Patel Gene: mdga1 has been classified as Red List (Low Evidence).
Mendeliome v1.4871 MDGA1 Chirag Patel gene: MDGA1 was added
gene: MDGA1 was added to Mendeliome. Sources: Literature
Mode of inheritance for gene: MDGA1 was set to BIALLELIC, autosomal or pseudoautosomal
Publications for gene: MDGA1 were set to 41862769; 40585099
Phenotypes for gene: MDGA1 were set to Neurodevelopmental disorder, MONDO:0700092, MDGA1-related
Review for gene: MDGA1 was set to AMBER
gene: MDGA1 was marked as current diagnostic
Added comment: 4 individuals from 2 unrelated families with biallelic missense variants in MDGA1 presenting with autism spectrum disorder, intellectual disability, and mild dysmorphic features. Functional assays in human hippocampal neurons demonstrate that the variants disrupt the triangular extracellular structure of the protein and cause loss-of-function in the negative regulation of GABAergic synapses. Additionally, Mdga1 male knock-in (KI) and conditional male knockout (cKO) mouse models recapitulate social and communicative deficits, with behavioral and electrophysiological deficits in male KI mice being rescued by Bazedoxifene. No behavioral deficits were seen in female counterparts. But authors state that extensive future validation in larger human cohorts, including functional studies of patient-derived cells, is needed to establish utility as a reliable diagnostic biomarker.
Sources: Literature