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Mendeliome v1.4336 NME5 Lucy Spencer Phenotypes for gene: NME5 were changed from Primary ciliary dyskinesia to Ciliary dyskinesia, primary, 48, without situs inversus MIM#620032
Mendeliome v1.4335 NME5 Lucy Spencer Publications for gene: NME5 were set to 32185794
Mendeliome v1.4334 NME5 Lucy Spencer Classified gene: NME5 as Green List (high evidence)
Mendeliome v1.4334 NME5 Lucy Spencer Gene: nme5 has been classified as Green List (High Evidence).
Mendeliome v1.4333 NME5 Lucy Spencer reviewed gene: NME5: Rating: GREEN; Mode of pathogenicity: None; Publications: 32185794, 33635012, 37296588, 37998386, 37957793, 41499646; Phenotypes: Ciliary dyskinesia, primary, 48, without situs inversus MIM#620032; Mode of inheritance: BIALLELIC, autosomal or pseudoautosomal
Mendeliome v0.3193 NME5 Zornitza Stark Marked gene: NME5 as ready
Mendeliome v0.3193 NME5 Zornitza Stark Gene: nme5 has been classified as Amber List (Moderate Evidence).
Mendeliome v0.3193 NME5 Zornitza Stark Classified gene: NME5 as Amber List (moderate evidence)
Mendeliome v0.3193 NME5 Zornitza Stark Gene: nme5 has been classified as Amber List (Moderate Evidence).
Mendeliome v0.3192 NME5 Zornitza Stark gene: NME5 was added
gene: NME5 was added to Mendeliome. Sources: Literature
Mode of inheritance for gene: NME5 was set to BIALLELIC, autosomal or pseudoautosomal
Publications for gene: NME5 were set to 32185794
Phenotypes for gene: NME5 were set to Primary ciliary dyskinesia
Review for gene: NME5 was set to AMBER
Added comment: One patient with PCD with situs solitus, with radial spokes (RS) and central pair (CP) defects. Patient had a homozygous nonsense variant in NME5, with parents as carriers. Morpholino knockdown of nme5 in zebrafish embryos resulted in motile cilia defects with phenotypes compatible with ciliopathy.
Sources: Literature