Activity

Filter

Cancel
Date Panel Item Activity
12 actions
Optic Atrophy v1.54 PPIB Zornitza Stark Tag founder tag was added to gene: PPIB.
Optic Atrophy v1.54 PPIB Zornitza Stark Marked gene: PPIB as ready
Optic Atrophy v1.54 PPIB Zornitza Stark Gene: ppib has been classified as Amber List (Moderate Evidence).
Optic Atrophy v1.54 PPIB Zornitza Stark Classified gene: PPIB as Amber List (moderate evidence)
Optic Atrophy v1.54 PPIB Zornitza Stark Gene: ppib has been classified as Amber List (Moderate Evidence).
Optic Atrophy v1.53 PPIB Zornitza Stark Classified gene: PPIB as Amber List (moderate evidence)
Optic Atrophy v1.53 PPIB Zornitza Stark Gene: ppib has been classified as Amber List (Moderate Evidence).
Optic Atrophy v1.52 PPIB Zornitza Stark Classified gene: PPIB as Red List (low evidence)
Optic Atrophy v1.52 PPIB Zornitza Stark Gene: ppib has been classified as Red List (Low Evidence).
Optic Atrophy v1.51 PPIB Zornitza Stark Classified gene: PPIB as Amber List (moderate evidence)
Optic Atrophy v1.51 PPIB Zornitza Stark Gene: ppib has been classified as Amber List (Moderate Evidence).
Optic Atrophy v1.50 PPIB Rylee Peters gene: PPIB was added
gene: PPIB was added to Optic Atrophy. Sources: Literature
Mode of inheritance for gene: PPIB was set to MONOALLELIC, autosomal or pseudoautosomal, NOT imprinted
Publications for gene: PPIB were set to 41045073
Phenotypes for gene: PPIB were set to Optic atrophy (MONDO:0003608), PPIB-related
Review for gene: PPIB was set to AMBER
Added comment: PMID: 41045073 report 19 individuals from 9 families with adult‑onset autosomal dominant optic atrophy with a recurrent p.(Arg180Trp) missense variant (present in 7 hets in gnomAD v4). Segregation testing also identified the variant in 7 unaffected individuals (6 of whom were younger than 30yo). Somalier (relatedness metric) found possible distant relationships between 3 families; and 5 families have a shared haplotype, indicating a possible founder effect. Patient-derived fibroblasts showed altered mitochondrial morphology and subtle respiratory chain defects.
Sources: Literature