Activity

Filter

Cancel
Date Panel Item Activity
15 actions
Skeletal dysplasia v0.394 TMEM251 Zornitza Stark changed review comment from: PMID 40171858: reports 2 siblings from an Iranian consanguineous family and six previously reported families (8 patients, 7 unrelated families) with biallelic loss‑of‑function LYSET variants presenting with MLII‑like mucolipidosis; core features include dysostosis multiplex, coarse facial features, hepatomegaly, joint contractures, developmental delay; mouse knockout recapitulates the phenotype, supporting gene‑disease causality.; to: PMID 40171858: reports 2 siblings from an Iranian consanguineous family and six previously reported families (8 patients, 7 unrelated families) with biallelic loss‑of‑function LYSET variants presenting with MLII‑like mucolipidosis; core features include dysostosis multiplex, coarse facial features, hepatomegaly, joint contractures, developmental delay; mouse knockout recapitulates the phenotype, supporting gene‑disease causality.

HGNC approved name is LYSET.
Skeletal dysplasia v0.394 TMEM251 Zornitza Stark Tag new gene name tag was added to gene: TMEM251.
Skeletal dysplasia v0.394 TMEM251 Zornitza Stark Publications for gene: TMEM251 were set to 33252156
Skeletal dysplasia v0.393 TMEM251 Zornitza Stark edited their review of gene: TMEM251: Changed publications: 40171858
Skeletal dysplasia v0.393 TMEM251 Zornitza Stark Classified gene: TMEM251 as Green List (high evidence)
Skeletal dysplasia v0.393 TMEM251 Zornitza Stark Gene: tmem251 has been classified as Green List (High Evidence).
Skeletal dysplasia v0.392 TMEM251 Zornitza Stark edited their review of gene: TMEM251: Changed rating: GREEN
Skeletal dysplasia v0.392 TMEM251 Zornitza Stark commented on gene: TMEM251: PMID 40171858: reports 2 siblings from an Iranian consanguineous family and six previously reported families (8 patients, 7 unrelated families) with biallelic loss‑of‑function LYSET variants presenting with MLII‑like mucolipidosis; core features include dysostosis multiplex, coarse facial features, hepatomegaly, joint contractures, developmental delay; mouse knockout recapitulates the phenotype, supporting gene‑disease causality.
Skeletal dysplasia v0.103 TMEM251 Zornitza Stark Phenotypes for gene: TMEM251 were changed from Dysostosis multiplex‐like skeletal dysplasia; severe short stature to Dysostosis multiplex, Ain-Naz type 619345
Skeletal dysplasia v0.102 TMEM251 Zornitza Stark reviewed gene: TMEM251: Rating: AMBER; Mode of pathogenicity: None; Publications: ; Phenotypes: Dysostosis multiplex, Ain-Naz type 619345; Mode of inheritance: BIALLELIC, autosomal or pseudoautosomal
Skeletal dysplasia v0.77 TMEM251 Bryony Thompson Marked gene: TMEM251 as ready
Skeletal dysplasia v0.77 TMEM251 Bryony Thompson Gene: tmem251 has been classified as Amber List (Moderate Evidence).
Skeletal dysplasia v0.77 TMEM251 Bryony Thompson Classified gene: TMEM251 as Amber List (moderate evidence)
Skeletal dysplasia v0.77 TMEM251 Bryony Thompson Gene: tmem251 has been classified as Amber List (Moderate Evidence).
Skeletal dysplasia v0.76 TMEM251 Bryony Thompson gene: TMEM251 was added
gene: TMEM251 was added to Skeletal dysplasia. Sources: Literature
Mode of inheritance for gene: TMEM251 was set to BIALLELIC, autosomal or pseudoautosomal
Publications for gene: TMEM251 were set to 33252156
Phenotypes for gene: TMEM251 were set to Dysostosis multiplex‐like skeletal dysplasia; severe short stature
Review for gene: TMEM251 was set to AMBER
Added comment: Two unrelated consanguineous families with homozygous variants (c.133C>T; p.Arg45Trp and c.215dupA; p.Tyr72Ter), with co-segregation data in one family. Preliminary in vitro functional assays conducted - Tmem251 knockdown by small interfering RNA induced dedifferentiation of rat primary chondrocytes.
Sources: Literature